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Old 07-06-2007, 11:50 PM
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Default Re: EPOC-alypse

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I'm not familiar with EPOC,

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It means excess post exercise oxygen consumption. Oxygen is an indirect measurement of caloric expenditure. If you are consuming more oxygen, it is infered that you are metabolizing more calories (about 5 calories per liter of oxygen consumed).

Right after exercise, EPOC is attributed to clearing wastes such as lactic acid, and prolonged effects of EPOC lasting up to 48 hours are attributed to increased thermogenic hormones such as T3, and catecholamines.

Here is a quote from the aforementioned article explaining.


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The Effect of Exercise Intensity on EPOC

It is well established that exercise increases oxygen consumption for several hours after its completion (Gaesser and Brooks, 1984). As discussed, oxygen consumption is used to assess caloric expenditure. Therefore elevated levels of 02 consumption reflect a higher resting metabolic rate. Explanations for such a phenomenon are connected to a number of historical events. It all began with Berzelius, who in 1808 found that lactate concentration was increased in ‘ the muscles of hunted stags( Gladden, 2004)’ who relied on anaerobic pathways to attempt to escape their predators. This was followed by Myeroff’s (1920) discovery that glycogen served as a precursor for lactate (Gladden, 2004). Building on this work, Hill proposed the 02 debt theory, which suggested that 1/5 of the increase in 02 consumption was used in the oxidation of lactate. This in turn provided the energy to convert lactate build up during exercise back to glycogen, thus repaying the ‘debt’ incurred through anaerobic processes. Scientists further noted that the 02 debt produced a curve that was characterized by a rapid phase of 02 dissipation, followed by a slow phase of decline. Margaria et al. (1933) called the fast phase alactacid, followed by the slower lactacid phase. The alactacid phase was postulated to account for replenishment of non lactic acid components of anaerobic energy utilization, such as the phosphorylation of free creatine to form creatine phosphate. The lactacid phase was said to replenish glycogen stores from lactate. However, Gaesser and Brooks (1984) suggested that these explanations were to simplistic and that evidence pointed to the majority of lactate being oxidized following exercise, with the remainder serving as a carbon skeleton for a number of processes of which glycogen replenishment is just one. Further, it was stated that the oxygen utilization could be linked to a number of phenomenon, including the residual effects of hormones, and increased temperature. In this historical review, Gaesser and Brooks (1984) introduced the new terms - excess post exercise oxygen consumption (EPOC) and recovery 02 to eliminate the ‘ implication of causality in describing the elevation in metabolic rate above resting levels after exercise.’

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