Researched and Composed by
Nicholas Igl
Introduction
As hardcore gym warriors we are constantly bombarded with obstacles aimed at
suppressing our bodies ability to grow. Many of these obstacles are easy to spot
and overcome; diet, protein intake, overtraining, etc. Others, are not so easy
to spot; central nervous system ( CNS ) adaptations, mitochondrial density
limitations,, etc etc.
In this article we will be taking a look at calcium. The well-known ( and
extremely abundant! ) mineral which rarely receives the attention it so
deserves.
Calcium is the most abundant mineral in our entire body. Calcium is responsible
for the regulation of our heart beat, blood pressure and water. It is also used
in blood clotting, the maintenance and formation of bones and teeth and the
conduction of electrical impulses in the nervous system.
However, as elite athletes we are more concerned with calcium’s ability to turn
on muscles! For us to fully understand this process we must go through exactly
how a muscle contraction is triggered. Here are the steps followed in all muscle
contractions:
- Muscle contraction is initiated from somewhere in the body's CNS, either
being initiated voluntarily from the brain or as a reflex instigated by the
spinal cord.
- A motor neuron in the spinal cord is activated and an action potential (
electrical signal ) transverses out.
- The axon branches to supply a number of muscle fibers called a motor unit,
and the action potential is conveyed to a motor end plate on each muscle
fiber.
- The action potential releases packets of acetylcholine into the synaptic
cleft on the surface of the muscle fiber. Acetylcholine is a chemical that
carries information across the synaptic cleft ( the space between two nerve
cells ).
- The action potential spreads inside the muscle fiber and enters the cell
through the T-tubule.
- The action potential opens the doors of the sarcoplasmic reticulum ( where
calcium is stored ).
- The now released calcium ions flow into the cytoplasm.
- Calcium ions bind to troponin-tropomyosin molecules which are located in
the grooves of actin filaments. When contraction is not about to take place
the tropomyosin covers the sites where actin and myosin can form crossbridges.
- The bound calcium ions cause the troponin to change shape and result in
the tropomyosin sliding out of the grooves, exposing the actin and myosin
sites.
- The myosin and actin reciprocate by cycling crossbridges. The muscle
shortens and force is a result.
As you can see, calcium plays a huge role in the triggering of a muscle
contraction. Without it our muscle simply could not function!
The majority of Americans do not get enough calcium in their diets. Now, being
bodybuilders we consume an adversely large amount of protein throughout the day.
High protein diets cause more calcium to be lost through urine. Also, Weight
training does strengthen bones, however, high lactic acid production inhibits
the re-absorption of calcium. And since we’re training “ beyond failure “ our
lactic acid production is sky high! This means that bodybuilders have an even
higher risk of becoming deficient!
Having a deficiency of calcium will greatly affect your performance in the
weight room and in all other parts of your life. Check out the symptoms of a
deficiency, some of them are not very pleasant:
- Muscle spasms and twitches
- Muscle cramps
- Nervousness
- Slow wound healing
- Insomnia
- Elevated cholesterol levels
- Brittle nails
- Heart palpitations ( irregular rapid beating )
- Aching joints
- High blood pressure
- Tooth decay
- Rheumatoid arthritis
- Irritability
By now I'm sure you're getting the point, you need to be supplementing with
calcium! But how much calcium do we really need? I recommend that you supplement
with 1000 milligrams of calcium carbonate or citrate ( the highest quality forms
) per day. However you can go all the way up to 2000 milligrams if you're a
larger bodybuilder producing large amounts of lactic acid, just make sure you
take it in two doses.
For the best absorption use a calcium supplement that contains both vitamin d
and magnesium as they both increase absorption and usage. Also, our large
protein consumptions also produce large amounts of metabolic waste, which raises
the acidity of our blood. This decreases our kidneys ability to reabsorb
calcium. We can work around this by consuming more vegetables and fiber
throughout the day. Fiber acts as a buffering agent which helps to lower the
acidity of our blood. Also, studies have shown that calcium is best absorbed
while sleeping. This means that you should take your calcium after the final
meal of the day as it contains large amounts of fiber and is taken just before
sleep. The only other method of supplementing with calcium that has been proven
is smaller doses spread throughout the entire day, which can become rather
tedious. However it can be more effective.
Conclusion
Novice bodybuilder or veteran, you should be supplementing with calcium.
Sincerely
" Nicholas Igl "
nick@abcbodybuilding.com
References
- Principles of Meat Science (4th Edition), chapter 4, pages 69 to
80.
- Barzel US, Massey LK. "Excess Dietary Protein Can Adversely Affect Bone"
J Nutri 1051-1053.
- Mete Civelek, Kristy Ainslie, Jeff S. Garanich, and John M. Tarbell.
"Smooth muscle cells contract in response to fluid flow via Ca2+-independent
signaling mechanism" J Appl Physiol 1907-1917.
- Amanda Devine, Scott G Wilson, Ian M Dick, and Richard L Prince. "Effects
of vitamin D metabolites on intestinal calcium absorption and bone turnover in
elderly women" Am J Clin Nutr 283-288.
- OK O'Brien, SA Abrams, LK Liang, KJ Ellis, and RF Gagel. "Increased
efficiency of calcium absorption during short periods of inadequate calcium
intake in girls" Am J Clin Nutr 579-583.
- H Spencer, L Kramer, D Osis, and C Norris. "Effect of a high protein
(meat) intake on calcium metabolism in man" Am J Clin Nutr 2167-2180.
- Merja UM Kärkkäinen, Christel JE Lamberg-Allardt, Suvi Ahonen, and Matti
Välimäki. "Does it make a difference how and when you take your calcium? The
acute effects of calcium on calcium and bone metabolism" Am J Clin Nutr
335-342.
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